I assume you're not seeing the linked story, sometimes posts propagate without them. I'm not sure how it works on whatever instance software you're using, but at least on MBin there's a button next to the community name in the sidebar which opens the community on the instance it originates from. The link is usually good there. In any event, the link is below:

https://case.edu/news/new-study-shows-alzheimers-disease-can-be-reversed-achieve-full-neurological-recovery-not-just-prevented-or-slowed-animal-models

[–] YetAnotherNerd@sopuli.xyz 2 points 3 months ago (1 children)

Yeah, just see the headline and the link to that site, that’s it. At least on Voyager.

[–] FeatherConstrictor@sh.itjust.works 3 points 3 months ago

Yah weird, usually crossposts render fine for me on Voyager but this one seems to have failed

[–] viral.vegabond@piefed.social 1 points 3 months ago

I could be wrong, but I seem to remember that one being an Mbin instance...

[–] solrize@lemmy.ml 5 points 3 months ago (1 children)

IN MICE

[–] MalReynolds@slrpnk.net 6 points 3 months ago

Which don't get Alzheimer's. Mice lie, monkeys exaggerate.

That said, this study is cause for cautious optimism. The mechanism they've targeted cleared symptoms in 'two of these mouse models: One carried multiple human mutations in amyloid processing; the other carried a human mutation in the tau protein. ' Mosreso, it does it by modulating NAD+, long a target of interest in AD. From the paper

Nicotinamide adenine dinucleotide (NAD+) homeostasis is central to cellular resilience against oxidative stress, DNA damage, neuroinflammation, blood-brain barrier (BBB) deterioration, impaired hippocampal neurogenesis, synaptic plasticity deficits, and neurodegeneration.

As such it is not only of interest to sufferers of AD but the aging population in general.